ALCOHOL Y EJERCICIO Tras el consumo agudo de alcohol, la gluconeogénesis hepática y la oxidación mitocondrial de glucosa y ácidos grasos se ven afectadas de manera rápida y significativa. El metabolismo del etanol incrementa el cociente NADH/NADtextsuperscript{ } en el citosol y la mitocondria, lo que inhibe la gluconeogénesis y la oxidación de ácidos grasos en el hígado. Estudios en modelos animales y humanos demuestran que estos efectos metabólicos pueden observarse en cuestión de horas tras l
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ALCOHOL Y EJERCICIO Tras el consumo agudo de alcohol, la gluconeogénesis hepática y la oxidación mitocondrial de glucosa y ácidos grasos se ven afectadas de manera rápida
Endocrinogustavoparra. . ALCOHOL Y EJERCICIO Tras el consumo agudo de alcohol, la gluconeogénesis hepática y la oxidación mitocondrial de glucosa y ácidos grasos se ven afectadas de manera rápida y significativa. El metabolismo del etanol incrementa el cociente NADH/NADtextsuperscript{ } en el citosol y la mitocondria, lo que inhibe la ...
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